"Pregnancy Proteins" is a descriptor in the National Library of Medicine's controlled vocabulary thesaurus,
MeSH (Medical Subject Headings). Descriptors are arranged in a hierarchical structure,
which enables searching at various levels of specificity.
Proteins produced by organs of the mother or the PLACENTA during PREGNANCY. These proteins may be pregnancy-specific (present only during pregnancy) or pregnancy-associated (present during pregnancy or under other conditions such as hormone therapy or certain malignancies.)
| Descriptor ID |
D011257
|
| MeSH Number(s) |
D12.776.780
|
| Concept/Terms |
Pregnancy Proteins- Pregnancy Proteins
- Proteins, Pregnancy
- Placental Proteins
- Proteins, Placental
|
Below are MeSH descriptors whose meaning is more general than "Pregnancy Proteins".
Below are MeSH descriptors whose meaning is more specific than "Pregnancy Proteins".
This graph shows the total number of publications written about "Pregnancy Proteins" by people in this website by year, and whether "Pregnancy Proteins" was a major or minor topic of these publications.
To see the data from this visualization as text,
click here.
| Year | Major Topic | Minor Topic | Total |
|---|
| 2008 | 0 | 1 | 1 |
| 2011 | 0 | 1 | 1 |
| 2014 | 1 | 0 | 1 |
| 2016 | 0 | 1 | 1 |
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Below are the most recent publications written about "Pregnancy Proteins" by people in Profiles.
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Liu M, Hassana S, Stiles JK. Heme-mediated apoptosis and fusion damage in BeWo trophoblast cells. Sci Rep. 2016 10 31; 6:36193.
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Wang X, Mendelsohn L, Rogers H, Leitman S, Raghavachari N, Yang Y, Yau YY, Tallack M, Perkins A, Taylor JG, Noguchi CT, Kato GJ. Heme-bound iron activates placenta growth factor in erythroid cells via erythroid Kr?ppel-like factor. Blood. 2014 Aug 07; 124(6):946-54.
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Bhat RK, Ellestad KK, Wheatley BM, Warren R, Holt RA, Power C. Age- and disease-dependent HERV-W envelope allelic variation in brain: association with neuroimmune gene expression. PLoS One. 2011 Apr 29; 6(4):e19176.
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Le XF, Mao W, Lu C, Thornton A, Heymach JV, Sood AK, Bast RC. Specific blockade of VEGF and HER2 pathways results in greater growth inhibition of breast cancer xenografts that overexpress HER2. Cell Cycle. 2008 Dec; 7(23):3747-58.