Nitric Oxide Donors

"Nitric Oxide Donors" is a descriptor in the National Library of Medicine's controlled vocabulary thesaurus, MeSH (Medical Subject Headings). Descriptors are arranged in a hierarchical structure, which enables searching at various levels of specificity.

MeSH information

Definition | Details | More General Concepts | Related Concepts | More Specific Concepts

A diverse group of agents, with unique chemical structures and biochemical requirements, which generate NITRIC OXIDE. These compounds have been used in the treatment of cardiovascular diseases and the management of acute myocardial infarction, acute and chronic congestive heart failure, and surgical control of blood pressure. (Adv Pharmacol 1995;34:361-81)

Descriptor ID	D020030
MeSH Number(s)	D27.505.519.656 D27.505.954.411.590
Concept/Terms	Nitric Oxide Donors Nitric Oxide Donors Donors, Nitric Oxide Oxide Donors, Nitric

Below are MeSH descriptors whose meaning is more general than "Nitric Oxide Donors".

Chemicals and Drugs [D]
Chemical Actions and Uses [D27]
Pharmacologic Actions [D27.505]
Molecular Mechanisms of Pharmacological Action [D27.505.519]
Nitric Oxide Donors [D27.505.519.656]
Therapeutic Uses [D27.505.954]
Cardiovascular Agents [D27.505.954.411]
Nitric Oxide Donors [D27.505.954.411.590]

Below are MeSH descriptors whose meaning is related to "Nitric Oxide Donors".

Below are MeSH descriptors whose meaning is more specific than "Nitric Oxide Donors".

publications

Timeline | Most Recent

This graph shows the total number of publications written about "Nitric Oxide Donors" by people in this website by year, and whether "Nitric Oxide Donors" was a major or minor topic of these publications.

Bar chart showing 13 publications over 6 distinct years, with a maximum of 4 publications in 2004

To see the data from this visualization as text, click here.

Year	Major Topic	Minor Topic	Total
2003	3	0	3
2004	1	3	4
2007	2	1	3
2011	0	1	1
2012	0	1	1
2016	0	1	1

Below are the most recent publications written about "Nitric Oxide Donors" by people in Profiles.

Martinez L, Thames E, Kim J, Chaudhuri G, Singh R, Pervin S. Increased sensitivity of African American triple negative breast cancer cells to nitric oxide-induced mitochondria-mediated apoptosis. BMC Cancer. 2016 07 29; 16:559.

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Crassous PA, Couloubaly S, Huang C, Zhou Z, Baskaran P, Kim DD, Papapetropoulos A, Fioramonti X, Dur?n WN, Beuve A. Soluble guanylyl cyclase is a target of angiotensin II-induced nitrosative stress in a hypertensive rat model. Am J Physiol Heart Circ Physiol. 2012 Sep 01; 303(5):H597-604.

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Ferrini MG, Moon J, Rivera S, Rajfer J, Gonzalez-Cadavid NF. Amelioration of diabetes-induced cavernosal fibrosis by antioxidant and anti-transforming growth factor-?1 therapies in inducible nitric oxide synthase-deficient mice. BJU Int. 2012 Feb; 109(4):586-93.

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Taylor AL, Ziesche S, Yancy CW, Carson P, Ferdinand K, Taylor M, Adams K, Olukotun AY, Ofili E, Tam SW, Sabolinski ML, Worcel M, Cohn JN. Early and sustained benefit on event-free survival and heart failure hospitalization from fixed-dose combination of isosorbide dinitrate/hydralazine: consistency across subgroups in the African-American Heart Failure Trial. Circulation. 2007 Apr 03; 115(13):1747-53.

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Lapu-Bula R, Ofili E. From hypertension to heart failure: role of nitric oxide-mediated endothelial dysfunction and emerging insights from myocardial contrast echocardiography. Am J Cardiol. 2007 Mar 26; 99(6B):7D-14D.

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Zhou G, Harruna II, Zhou WL, Aicher WK, Geckeler KE. Nanostructured thermosensitive polymers with radical scavenging ability. Chemistry. 2007; 13(2):569-73.

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Browner NC, Dey NB, Bloch KD, Lincoln TM. Regulation of cGMP-dependent protein kinase expression by soluble guanylyl cyclase in vascular smooth muscle cells. J Biol Chem. 2004 Nov 05; 279(45):46631-6.

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Aliyev A, Seyidova D, Rzayev N, Obrenovich ME, Lamb BT, Chen SG, Smith MA, Perry G, de la Torre JC, Aliev G. Is nitric oxide a key target in the pathogenesis of brain lesions during the development of Alzheimer's disease? Neurol Res. 2004 Jul; 26(5):547-53.

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Qin Q, Yang XM, Cui L, Critz SD, Cohen MV, Browner NC, Lincoln TM, Downey JM. Exogenous NO triggers preconditioning via a cGMP- and mitoKATP-dependent mechanism. Am J Physiol Heart Circ Physiol. 2004 Aug; 287(2):H712-8.

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Browner NC, Sellak H, Lincoln TM. Downregulation of cGMP-dependent protein kinase expression by inflammatory cytokines in vascular smooth muscle cells. Am J Physiol Cell Physiol. 2004 Jul; 287(1):C88-96.

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