Cyclic AMP Response Element-Binding Protein

"Cyclic AMP Response Element-Binding Protein" is a descriptor in the National Library of Medicine's controlled vocabulary thesaurus, MeSH (Medical Subject Headings). Descriptors are arranged in a hierarchical structure, which enables searching at various levels of specificity.

MeSH information

Definition | Details | More General Concepts | Related Concepts | More Specific Concepts

A protein that has been shown to function as a calcium-regulated transcription factor as well as a substrate for depolarization-activated CALCIUM-CALMODULIN-DEPENDENT PROTEIN KINASES. This protein functions to integrate both calcium and cAMP signals.

Descriptor ID	D017362
MeSH Number(s)	D12.776.260.108.184 D12.776.930.127.184
Concept/Terms	Cyclic AMP Response Element-Binding Protein Cyclic AMP Response Element-Binding Protein CREB Protein DNA-Binding Protein, Cyclic AMP-Responsive DNA Binding Protein, Cyclic AMP Responsive cAMP Response Element-Binding Protein cAMP Response Element Binding Protein Cyclic AMP-Responsive DNA-Binding Protein Cyclic AMP Responsive DNA Binding Protein

Below are MeSH descriptors whose meaning is more general than "Cyclic AMP Response Element-Binding Protein".

Chemicals and Drugs [D]
Amino Acids, Peptides, and Proteins [D12]
Proteins [D12.776]
DNA-Binding Proteins [D12.776.260]
Basic-Leucine Zipper Transcription Factors [D12.776.260.108]
Cyclic AMP Response Element-Binding Protein [D12.776.260.108.184]
Transcription Factors [D12.776.930]
Basic-Leucine Zipper Transcription Factors [D12.776.930.127]
Cyclic AMP Response Element-Binding Protein [D12.776.930.127.184]

Below are MeSH descriptors whose meaning is related to "Cyclic AMP Response Element-Binding Protein".

Below are MeSH descriptors whose meaning is more specific than "Cyclic AMP Response Element-Binding Protein".

publications

Timeline | Most Recent

This graph shows the total number of publications written about "Cyclic AMP Response Element-Binding Protein" by people in this website by year, and whether "Cyclic AMP Response Element-Binding Protein" was a major or minor topic of these publications.

Bar chart showing 36 publications over 19 distinct years, with a maximum of 5 publications in 2005

To see the data from this visualization as text, click here.

Year	Major Topic	Minor Topic	Total
1999	1	0	1
2001	1	2	3
2003	1	0	1
2004	0	1	1
2005	2	3	5
2006	0	1	1
2007	1	0	1
2008	1	0	1
2009	1	2	3
2011	0	1	1
2012	0	1	1
2013	2	2	4
2014	2	1	3
2015	0	4	4
2017	0	1	1
2018	2	0	2
2019	0	1	1
2020	1	0	1
2021	0	1	1

Below are the most recent publications written about "Cyclic AMP Response Element-Binding Protein" by people in Profiles.

Yan J, Xu W, Lenahan C, Huang L, Wen J, Li G, Hu X, Zheng W, Zhang JH, Tang J. CCR5 Activation Promotes NLRP1-Dependent Neuronal Pyroptosis via CCR5/PKA/CREB Pathway After Intracerebral Hemorrhage. Stroke. 2021 12; 52(12):4021-4032.

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Dai W, Xu Y, Mo S, Li Q, Yu J, Wang R, Ma Y, Ni Y, Xiang W, Han L, Zhang L, Cai S, Qin J, Chen WL, Jia W, Cai G. GLUT3 induced by AMPK/CREB1 axis is key for withstanding energy stress and augments the efficacy of current colorectal cancer therapies. Signal Transduct Target Ther. 2020 09 02; 5(1):177.

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Zhao Y, Kang X, Gao F, Guzman A, Lau RP, Biniwale R, Wadehra M, Reemtsen B, Garg M, Halnon N, Quintero-Rivera F, Van Arsdell G, Coppola G, Nelson SF, Touma M. Gene-environment regulatory circuits of right ventricular pathology in tetralogy of fallot. J Mol Med (Berl). 2019 12; 97(12):1711-1722.

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Feldmann KG, Chowdhury A, Becker JL, McAlpin N, Ahmed T, Haider S, Richard Xia JX, Diaz K, Mehta MG, Mano I. Non-canonical activation of CREB mediates neuroprotection in a Caenorhabditis elegans model of excitotoxic necrosis. J Neurochem. 2019 02; 148(4):531-549.

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Mo J, Enkhjargal B, Travis ZD, Zhou K, Wu P, Zhang G, Zhu Q, Zhang T, Peng J, Xu W, Ocak U, Chen Y, Tang J, Zhang J, Zhang JH. AVE 0991 attenuates oxidative stress and neuronal apoptosis via Mas/PKA/CREB/UCP-2 pathway after subarachnoid hemorrhage in rats. Redox Biol. 2019 01; 20:75-86.

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Montoya A, Amaya CN, Belmont A, Diab N, Trevino R, Villanueva G, Rains S, Sanchez LA, Badri N, Otoukesh S, Khammanivong A, Liss D, Baca ST, Aguilera RJ, Dickerson EB, Torabi A, Dwivedi AK, Abbas A, Chambers K, Bryan BA, Nahleh Z. Use of non-selective ?-blockers is associated with decreased tumor proliferative indices in early stage breast cancer. Oncotarget. 2017 Jan 24; 8(4):6446-6460.

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Dao AT, Zagaar MA, Levine AT, Alkadhi KA. Comparison of the Effect of Exercise on Late-Phase LTP of the Dentate Gyrus and CA1 of Alzheimer's Disease Model. Mol Neurobiol. 2016 12; 53(10):6859-6868.

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Torres OV, McCoy MT, Ladenheim B, Jayanthi S, Brannock C, Tulloch I, Krasnova IN, Cadet JL. CAMKII-conditional deletion of histone deacetylase 2 potentiates acute methamphetamine-induced expression of immediate early genes in the mouse nucleus accumbens. Sci Rep. 2015 Aug 24; 5:13396.

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Wu Y, Sarkissyan M, Mcghee E, Lee S, Vadgama JV. Combined inhibition of glycolysis and AMPK induces synergistic breast cancer cell killing. Breast Cancer Res Treat. 2015 Jun; 151(3):529-39.

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Zagaar MA, Dao AT, Alhaider IA, Alkadhi KA. Prevention by Regular Exercise of Acute Sleep Deprivation-Induced Impairment of Late Phase LTP and Related Signaling Molecules in the Dentate Gyrus. Mol Neurobiol. 2016 07; 53(5):2900-2910.

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