The Francisella tularensis pathogenicity island protein IglC and its regulator MglA are essential for modulating phagosome biogenesis and subsequent bacterial escape into the cytoplasm.

Santic M, Molmeret M, Klose KE, Jones S, Kwaik YA. The Francisella tularensis pathogenicity island protein IglC and its regulator MglA are essential for modulating phagosome biogenesis and subsequent bacterial escape into the cytoplasm. Cell Microbiol. 2005 Jul; 7(7):969-79.

View in: PubMed

subject areas

Antigens, CD
Bacterial Proteins
Cathepsin D
Cells, Cultured
Cytoplasm
Francisella tularensis
Genomic Islands
Humans
Lysosomes
Macrophages
Mutation
Phagosomes
Virulence Factors

authors with profiles

Karl E. Klose

RCMI CC is supported by the National Institute on Minority Health and Health Disparities, National Institutes of Health (NIH), through Grant Number U24MD015970. The contents of this site are solely the responsibility of the authors and do not necessarily represent the official views of the NIH

For technical support please contact support