Inclusion Bodies

"Inclusion Bodies" is a descriptor in the National Library of Medicine's controlled vocabulary thesaurus, MeSH (Medical Subject Headings). Descriptors are arranged in a hierarchical structure, which enables searching at various levels of specificity.

MeSH information

Definition | Details | More General Concepts | Related Concepts | More Specific Concepts

A generic term for any circumscribed mass of foreign (e.g., lead or viruses) or metabolically inactive materials (e.g., ceroid or MALLORY BODIES), within the cytoplasm or nucleus of a cell. Inclusion bodies are in cells infected with certain filtrable viruses, observed especially in nerve, epithelial, or endothelial cells. (Stedman, 25th ed)

Descriptor ID	D002479
MeSH Number(s)	A11.284.420
Concept/Terms	Inclusion Bodies Inclusion Bodies Bodies, Inclusion Body, Inclusion Inclusion Body Cellular Inclusions Cellular Inclusion Inclusion, Cellular Inclusions, Cellular Cytoplasmic Inclusions Cytoplasmic Inclusions Cytoplasmic Inclusion Inclusion, Cytoplasmic Inclusions, Cytoplasmic

Below are MeSH descriptors whose meaning is more general than "Inclusion Bodies".

Anatomy [A]
Cells [A11]
Cellular Structures [A11.284]
Inclusion Bodies [A11.284.420]

Below are MeSH descriptors whose meaning is related to "Inclusion Bodies".

Below are MeSH descriptors whose meaning is more specific than "Inclusion Bodies".

publications

Timeline | Most Recent

This graph shows the total number of publications written about "Inclusion Bodies" by people in this website by year, and whether "Inclusion Bodies" was a major or minor topic of these publications.

Bar chart showing 24 publications over 17 distinct years, with a maximum of 3 publications in 2003 and 2018

To see the data from this visualization as text, click here.

Year	Major Topic	Minor Topic	Total
1994	1	0	1
1996	1	0	1
1997	2	0	2
1998	1	0	1
2001	0	1	1
2003	1	2	3
2004	0	1	1
2005	0	1	1
2006	1	1	2
2008	1	0	1
2009	0	1	1
2010	1	1	2
2011	1	0	1
2014	0	1	1
2015	1	0	1
2018	1	2	3
2020	0	1	1

Below are the most recent publications written about "Inclusion Bodies" by people in Profiles.

Igietseme JU, Partin J, George Z, Omosun Y, Goldstein J, Joseph K, Ellerson D, Eko FO, Pohl J, Bandea C, Black CM. Epidermal Growth Factor Receptor and Transforming Growth Factor ? Signaling Pathways Cooperate To Mediate Chlamydia Pathogenesis. Infect Immun. 2020 03 23; 88(4).

View in: PubMed
George Z, Omosun Y, Azenabor AA, Goldstein J, Partin J, Joseph K, Ellerson D, He Q, Eko F, McDonald MA, Reed M, Svoboda P, Stuchlik O, Pohl J, Lutter E, Bandea C, Black CM, Igietseme JU. The molecular mechanism of induction of unfolded protein response by Chlamydia. Biochem Biophys Res Commun. 2019 01 08; 508(2):421-429.

View in: PubMed
Ramos-Ben?tez MJ, Lopez-Cruz LM, Aguayo V, Ruiz-Jimenez C, Espino AM. Cell-free expression, purification and immunoreactivity assessment of recombinant Fasciola hepatica saposin-like protein-2. Mol Biol Rep. 2018 Oct; 45(5):1551-1556.

View in: PubMed
Richard CA, Rincheval V, Lassoued S, Fix J, Cardone C, Esneau C, Nekhai S, Galloux M, Rameix-Welti MA, Sizun C, El?ou?t JF. RSV hijacks cellular protein phosphatase 1 to regulate M2-1 phosphorylation and viral transcription. PLoS Pathog. 2018 03; 14(3):e1006920.

View in: PubMed
Rivera F, Espino AM. Adjuvant-enhanced antibody and cellular responses to inclusion bodies expressing FhSAP2 correlates with protection of mice to Fasciola hepatica. Exp Parasitol. 2016 Jan; 160:31-8.

View in: PubMed
Tran LT, Gentil BJ, Sullivan KE, Durham HD. The voltage-gated calcium channel blocker lomerizine is neuroprotective in motor neurons expressing mutant SOD1, but not TDP-43. J Neurochem. 2014 Aug; 130(3):455-66.

View in: PubMed
Pereira MG, Nakayasu ES, Sant'Anna C, De Cicco NN, Atella GC, de Souza W, Almeida IC, Cunha-e-Silva N. Trypanosoma cruzi epimastigotes are able to store and mobilize high amounts of cholesterol in reservosome lipid inclusions. PLoS One. 2011; 6(7):e22359.

View in: PubMed
Heath JE, Siedlak SL, Zhu X, Lee HG, Thakur A, Yan R, Perry G, Smith MA, Castellani RJ. Widespread distribution of reticulon-3 in various neurodegenerative diseases. Neuropathology. 2010 Dec; 30(6):574-9.

View in: PubMed
Clausen TH, Lamark T, Isakson P, Finley K, Larsen KB, Brech A, ?vervatn A, Stenmark H, Bj?rk?y G, Simonsen A, Johansen T. p62/SQSTM1 and ALFY interact to facilitate the formation of p62 bodies/ALIS and their degradation by autophagy. Autophagy. 2010 Apr; 6(3):330-44.

View in: PubMed
Pierre SR, Lemmens MA, Figueiredo-Pereira ME. Subchronic infusion of the product of inflammation prostaglandin J2 models sporadic Parkinson's disease in mice. J Neuroinflammation. 2009 Jul 25; 6:18.

View in: PubMed

People

Explore

Similar Concepts

Top Journals