"Receptors, IgE" is a descriptor in the National Library of Medicine's controlled vocabulary thesaurus,
MeSH (Medical Subject Headings). Descriptors are arranged in a hierarchical structure,
which enables searching at various levels of specificity.
Specific molecular sites on the surface of B- and T-lymphocytes which combine with IgEs. Two subclasses exist: low affinity receptors (Fc epsilon RII) and high affinity receptors (Fc epsilon RI).
Descriptor ID |
D017455
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MeSH Number(s) |
D12.776.543.750.705.871.280
|
Concept/Terms |
Receptors, IgE- Receptors, IgE
- CD23 Antigen
- Antigen, CD23
- Antigens, CD23
- CD23 Antigens
- IgE Receptors
- Immunoglobulin E Receptor
- Receptor, Immunoglobulin E
- Fc Receptors, epsilon
- Receptors, epsilon Fc
- epsilon Fc Receptors
- Fc epsilon Receptors
- epsilon Receptors, Fc
- Receptors, Fc epsilon
- CD 23 Antigens
- Antigens, CD 23
|
Below are MeSH descriptors whose meaning is more general than "Receptors, IgE".
Below are MeSH descriptors whose meaning is more specific than "Receptors, IgE".
This graph shows the total number of publications written about "Receptors, IgE" by people in this website by year, and whether "Receptors, IgE" was a major or minor topic of these publications.
To see the data from this visualization as text,
click here.
Year | Major Topic | Minor Topic | Total |
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2000 | 0 | 1 | 1 |
2002 | 1 | 0 | 1 |
2003 | 1 | 0 | 1 |
2004 | 1 | 0 | 1 |
2005 | 2 | 1 | 3 |
2007 | 1 | 0 | 1 |
2008 | 2 | 0 | 2 |
2009 | 0 | 1 | 1 |
2011 | 1 | 0 | 1 |
2012 | 0 | 1 | 1 |
2014 | 1 | 0 | 1 |
2016 | 1 | 0 | 1 |
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click here.
Below are the most recent publications written about "Receptors, IgE" by people in Profiles.
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Rodriguez AR, Yu JJ, Navara C, Chambers JP, Guentzel MN, Arulanandam BP. Contribution of Fc?RI-associated vesicles to mast cell-macrophage communication following Francisella tularensis infection. Innate Immun. 2016 10; 22(7):567-74.
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Umemoto EY, Speck M, Shimoda LM, Kahue K, Sung C, Stokes AJ, Turner H. Single-walled carbon nanotube exposure induces membrane rearrangement and suppression of receptor-mediated signalling pathways in model mast cells. Toxicol Lett. 2014 Aug 17; 229(1):198-209.
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Hunter C, Rodriguez A, Yu JJ, Chambers J, Guentzel MN, Arulanandam B. Comparison of bone marrow-derived and mucosal mast cells in controlling intramacrophage Francisella tularensis replication. Exp Biol Med (Maywood). 2012 Jun; 237(6):617-21.
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Thathiah P, Sanapala S, Rodriguez AR, Yu JJ, Murthy AK, Guentzel MN, Forsthuber TG, Chambers JP, Arulanandam BP. Non-FceR bearing mast cells secrete sufficient interleukin-4 to control Francisella tularensis replication within macrophages. Cytokine. 2011 Aug; 55(2):211-20.
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Fifadara NH, Aye CC, Raghuwanshi SK, Richardson RM, Ono SJ. CCR1 expression and signal transduction by murine BMMC results in secretion of TNF-alpha, TGFbeta-1 and IL-6. Int Immunol. 2009 Aug; 21(8):991-1001.
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Matsui T, Nunomura S, Shimokawa T, Yoshimaru T, Ra C. Functionality of the IgA Fc receptor (FcalphaR, CD89) is down-regulated by extensive engagement of FcepsilonRI. Clin Immunol. 2008 Oct; 129(1):155-62.
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Kashyap M, Thornton AM, Norton SK, Barnstein B, Macey M, Brenzovich J, Shevach E, Leonard WJ, Ryan JJ. Cutting edge: CD4 T cell-mast cell interactions alter IgE receptor expression and signaling. J Immunol. 2008 Feb 15; 180(4):2039-43.
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Beer F, Kuo CH, Morohoshi K, Goodliffe J, Munro P, Aye CC, Dawson M, Richardson RM, Jones LH, Ikeda Y, Nakamura T, Toda M, Flynn T, Ohbayashi M, Miyazaki D, Ono SJ. Role of beta-chemokines in mast cell activation and type I hypersensitivity reactions in the conjunctiva: in vivo and in vitro studies. Immunol Rev. 2007 Jun; 217:96-104.
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Stokes AJ, Shimoda LM, Lee JW, Rillero C, Chang YT, Turner H. Fcepsilon RI control of Ras via inositol (1,4,5) trisphosphate 3-kinase and inositol tetrakisphosphate. Cell Signal. 2006 May; 18(5):640-51.
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Miyazaki D, Nakamura T, Toda M, Cheung-Chau KW, Richardson RM, Ono SJ. Macrophage inflammatory protein-1alpha as a costimulatory signal for mast cell-mediated immediate hypersensitivity reactions. J Clin Invest. 2005 Feb; 115(2):434-42.