Thiol oxidative stress induced by metabolic disorders amplifies macrophage chemotactic responses and accelerates atherogenesis and kidney injury in LDL receptor-deficient mice.

Qiao M, Zhao Q, Lee CF, Tannock LR, Smart EJ, LeBaron RG, Phelix CF, Rangel Y, Asmis R. Thiol oxidative stress induced by metabolic disorders amplifies macrophage chemotactic responses and accelerates atherogenesis and kidney injury in LDL receptor-deficient mice. Arterioscler Thromb Vasc Biol. 2009 Nov; 29(11):1779-86.

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subject areas

Analysis of Variance
Animals
Atherosclerosis
Blood Chemical Analysis
Cells, Cultured
Chemotaxis
Diabetes Mellitus, Experimental
Disease Models, Animal
Female
Hypercholesterolemia
Kidney Diseases
Macrophages, Peritoneal
Mice
Mice, Inbred C57BL
Mice, Transgenic
Oxidation-Reduction
Oxidative Stress
Probability
Random Allocation
Receptors, LDL
Streptozocin
Sulfhydryl Compounds
Urinalysis

authors with profiles

Richard LeBaron
Clyde Phelix