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Michelle Juarez
Title Researcher
Faculty Rank Assistant Medical Professor
Degree Ph.D.
Institution City College, CUNY
Department Sophie Davis Program of Biomedical Education
Clusters Cancer
Infectious and Immunological Diseases
Address
160 Convent Ave.
Marshak Rm-510
City New York
State NY
Postal Code 10031
Telephone 212-650-7715
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2015 NIH NIAID 1R03AI117671

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  Genetic and molecular analyses of epidermal wound response during injury in Drosophila. Overall goal is to use the intersection between epidermal wound response and innate immunity to improve repair strategies in humans. Additional projects in Science Communication and STEM Outreach.

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BIOMEDICAL RESEARCH BIOMEDICAL - BASIC
BIOMEDICAL - BASIC CELL BIOLOGY
BIOMEDICAL - BASIC DEVELOPMENTAL BIOLOGY/EMBRYOLOGY
BIOMEDICAL - BASIC GENETICS, HUMAN AND ANIMAL

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1. Juarez MT, Kenet CM. Translating Research as an Approach to Enhance Science Engagement. Int J Environ Res Public Health. 2018 Aug 15; 15(8).
2. Capilla A, Karachentsev D, Patterson RA, Hermann A, Juarez MT, McGinnis W. Toll pathway is required for wound-induced expression of barrier repair genes in the Drosophila epidermis. Proc Natl Acad Sci U S A. 2017 03 28; 114(13):E2682-E2688.
3. Juarez MT, Kenet CM, Johnson CN. Communicating Science through a Novel Type of Journal. CBE Life Sci Educ. 2017; 16(2).
4. Juarez MT. Drosophila Embryos as a Model for Wound-Induced Transcriptional Dynamics: Genetic Strategies to Achieve a Localized Wound Response. Adv Wound Care (New Rochelle). 2016 Jun 01; 5(6):262-270.
5. Juarez MT, Patterson RA, Li W, McGinnis W. Microinjection wound assay and in vivo localization of epidermal wound response reporters in Drosophila embryos. J Vis Exp. 2013 Nov 01; (81):e50750.
6. Patterson RA, Juarez MT, Hermann A, Sasik R, Hardiman G, McGinnis W. Serine proteolytic pathway activation reveals an expanded ensemble of wound response genes in Drosophila. PLoS One. 2013; 8(4):e61773.
7. Paré A, Kim M, Juarez MT, Brody S, McGinnis W. The functions of grainy head-like proteins in animals and fungi and the evolution of apical extracellular barriers. PLoS One. 2012; 7(5):e36254.
8. Juarez MT, Patterson RA, Sandoval-Guillen E, McGinnis W. Duox, Flotillin-2, and Src42A are required to activate or delimit the spread of the transcriptional response to epidermal wounds in Drosophila. PLoS Genet. 2011 Dec; 7(12):e1002424.
9. Pearson JC, Juarez MT, Kim M, Drivenes Ø, McGinnis W. Multiple transcription factor codes activate epidermal wound-response genes in Drosophila. Proc Natl Acad Sci U S A. 2009 Feb 17; 106(7):2224-9.
10. Nogueira FT, Madi S, Chitwood DH, Juarez MT, Timmermans MC. Two small regulatory RNAs establish opposing fates of a developmental axis. Genes Dev. 2007 Apr 01; 21(7):750-5.
11. Juarez MT, Twigg RW, Timmermans MC. Specification of adaxial cell fate during maize leaf development. Development. 2004 Sep; 131(18):4533-44.
12. Juarez MT, Kui JS, Thomas J, Heller BA, Timmermans MC. microRNA-mediated repression of rolled leaf1 specifies maize leaf polarity. Nature. 2004 Mar 04; 428(6978):84-8.
13. Timmermans MC, Juarez MT, Phelps-Durr TL. A conserved microRNA signal specifies leaf polarity. Cold Spring Harb Symp Quant Biol. 2004; 69:409-17.
14. Ori N, Juarez MT, Jackson D, Yamaguchi J, Banowetz GM, Hake S. Leaf senescence is delayed in tobacco plants expressing the maize homeobox gene knotted1 under the control of a senescence-activated promoter. Plant Cell. 1999 Jun; 11(6):1073-80.

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Keywords
Wound Healing
Epidermis
Zea mays
Science
Plant Leaves
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