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One or more keywords matched the following items that are connected to Foster, David A.
Item TypeName
Academic Article Suppression of hypoxia-inducible factor 2alpha restores p53 activity via Hdm2 and reverses chemoresistance of renal carcinoma cells.
Academic Article Phospholipase D stabilizes HDM2 through an mTORC2/SGK1 pathway.
Academic Article Suppression of AKT phosphorylation restores rapamycin-based synthetic lethality in SMAD4-defective pancreatic cancer cells.
Academic Article High-dose rapamycin induces apoptosis in human cancer cells by dissociating mTOR complex 1 and suppressing phosphorylation of 4E-BP1.
Academic Article Inhibition of fatty acid synthase induces pro-survival Akt and ERK signaling in K-Ras-driven cancer cells.
Academic Article Apoptotic effects of high-dose rapamycin occur in S-phase of the cell cycle.
Academic Article Aspartate Rescues S-phase Arrest Caused by Suppression of Glutamine Utilization in KRas-driven Cancer Cells.
Academic Article Lipid sensing by mTOR complexes via de novo synthesis of phosphatidic acid.
Academic Article Glutamine as an Essential Amino Acid for KRas-Driven Cancer Cells.
Academic Article Phospholipase D overcomes cell cycle arrest induced by high-intensity Raf signaling.
Academic Article Phospholipase D elevates the level of MDM2 and suppresses DNA damage-induced increases in p53.
Academic Article Survival signals generated by estrogen and phospholipase D in MCF-7 breast cancer cells are dependent on Myc.
Academic Article Myc stabilization in response to estrogen and phospholipase D in MCF-7 breast cancer cells.
Academic Article Differential dependence of hypoxia-inducible factors 1 alpha and 2 alpha on mTORC1 and mTORC2.
Academic Article Regulation of mTORC1 and mTORC2 complex assembly by phosphatidic acid: competition with rapamycin.
Concept Proto-Oncogene Proteins
Concept Proto-Oncogene Proteins c-myc
Concept Proto-Oncogene Proteins p21(ras)
Concept Proto-Oncogene Proteins c-raf
Concept Proto-Oncogene Proteins c-akt
Concept Proto-Oncogene Proteins c-mdm2
Academic Article Inhibiting glutamine utilization creates a synthetic lethality for suppression of ATP citrate lyase in KRas-driven cancer cells.
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  • Proto Oncogenes
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