"Rats, Inbred Dahl" is a descriptor in the National Library of Medicine's controlled vocabulary thesaurus,
MeSH (Medical Subject Headings). Descriptors are arranged in a hierarchical structure,
which enables searching at various levels of specificity.
Inbred rats derived from Sprague-Dawley rats and used for the study of salt-dependent hypertension. Salt-sensitive and salt-resistant strains have been selectively bred to show the opposite genetically determined blood pressure responses to excess sodium chloride ingestion.
| Descriptor ID |
D020303
|
| MeSH Number(s) |
B01.050.050.199.520.760.165 B01.050.150.900.649.313.992.635.505.700.400.165
|
| Concept/Terms |
Rats, Inbred Dahl- Rats, Inbred Dahl
- Dahl Rats, Inbred
- Inbred Dahl Rats
- Dahl Rats
- Rats, Dahl
Dahl Salt-Resistant Rats- Dahl Salt-Resistant Rats
- Dahl Salt Resistant Rats
- Rats, Dahl Salt-Resistant
- Salt-Resistant Rats, Dahl
Dahl Salt-Sensitive Rats- Dahl Salt-Sensitive Rats
- Dahl Salt Sensitive Rats
- Rats, Dahl Salt-Sensitive
- Salt-Sensitive Rats, Dahl
- Dahl Hypertensive Rats
- Hypertensive Rats, Dahl
- Rats, Dahl Hypertensive
|
Below are MeSH descriptors whose meaning is more general than "Rats, Inbred Dahl".
Below are MeSH descriptors whose meaning is more specific than "Rats, Inbred Dahl".
This graph shows the total number of publications written about "Rats, Inbred Dahl" by people in this website by year, and whether "Rats, Inbred Dahl" was a major or minor topic of these publications.
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| Year | Major Topic | Minor Topic | Total |
|---|
| 2002 | 1 | 2 | 3 |
| 2003 | 0 | 3 | 3 |
| 2004 | 1 | 2 | 3 |
| 2005 | 1 | 4 | 5 |
| 2006 | 0 | 1 | 1 |
| 2007 | 0 | 1 | 1 |
| 2009 | 0 | 1 | 1 |
| 2010 | 0 | 1 | 1 |
| 2011 | 0 | 1 | 1 |
| 2012 | 0 | 1 | 1 |
| 2017 | 0 | 1 | 1 |
| 2019 | 0 | 1 | 1 |
| 2022 | 0 | 1 | 1 |
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Below are the most recent publications written about "Rats, Inbred Dahl" by people in Profiles.
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Bridges LE, Williams CL, Awumey EM. High Salt Upregulates Ca2+-Sensing Receptor Expression and Ca2+-Induced Relaxation of Contracted Mesenteric Arteries from Dahl Salt-Sensitive Rats. J Pharmacol Exp Ther. 2022 05; 381(2):120-128.
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Odutola SO, Bridges LE, Awumey EM. Protein Kinase C Downregulation Enhanced Extracellular Ca2+-Induced Relaxation of Isolated Mesenteric Arteries from Aged Dahl Salt-Sensitive Rats. J Pharmacol Exp Ther. 2019 09; 370(3):427-435.
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Zhou Y, Castonguay P, Sidhom EH, Clark AR, Dvela-Levitt M, Kim S, Sieber J, Wieder N, Jung JY, Andreeva S, Reichardt J, Dubois F, Hoffmann SC, Basgen JM, Montesinos MS, Weins A, Johnson AC, Lander ES, Garrett MR, Hopkins CR, Greka A. A small-molecule inhibitor of TRPC5 ion channels suppresses progressive kidney disease in animal models. Science. 2017 12 08; 358(6368):1332-1336.
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Bayorh M, Rollins-Hairston A, Adiyiah J, Lyn D, Eatman D. Eplerenone inhibits aldosterone-induced renal expression of cyclooxygenase. J Renin Angiotensin Aldosterone Syst. 2012 Sep; 13(3):353-9.
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Bayorh MA, Rollins-Hairston A, Adiyiah J, Lyn D, Eatman D. Eplerenone suppresses aldosterone/ salt-induced expression of NOX-4. J Renin Angiotensin Aldosterone Syst. 2011 Sep; 12(3):195-201.
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Eatman D, Layas MF, Bayorh MA. Eplerenone suppresses salt-induced vascular endothelial growth factor expression in the kidney. Kidney Blood Press Res. 2010; 33(3):167-73.
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Chandramohan G, Durham N, Sinha S, Norris K, Vaziri ND. Role of gamma melanocyte-stimulating hormone-renal melanocortin 3 receptor system in blood pressure regulation in salt-resistant and salt-sensitive rats. Metabolism. 2009 Oct; 58(10):1424-9.
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Chandramohan G, Bai Y, Norris K, Rodriguez-Iturbe B, Vaziri ND. Effects of dietary salt on intrarenal angiotensin system, NAD(P)H oxidase, COX-2, MCP-1 and PAI-1 expressions and NF-kappaB activity in salt-sensitive and -resistant rat kidneys. Am J Nephrol. 2008; 28(1):158-67.
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Bayorh MA, Mann G, Walton M, Eatman D. Effects of enalapril, tempol, and eplerenone on salt-induced hypertension in dahl salt-sensitive rats. Clin Exp Hypertens. 2006 Feb; 28(2):121-32.
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Bayorh MA, Ganafa AA, Eatman D, Walton M, Feuerstein GZ. Simvastatin and losartan enhance nitric oxide and reduce oxidative stress in salt-induced hypertension. Am J Hypertens. 2005 Nov; 18(11):1496-502.