"Neurocysticercosis" is a descriptor in the National Library of Medicine's controlled vocabulary thesaurus,
MeSH (Medical Subject Headings). Descriptors are arranged in a hierarchical structure,
which enables searching at various levels of specificity.
Infection of the brain, spinal cord, or perimeningeal structures with the larval forms of the genus TAENIA (primarily T. solium in humans). Lesions formed by the organism are referred to as cysticerci. The infection may be subacute or chronic, and the severity of symptoms depends on the severity of the host immune response and the location and number of lesions. SEIZURES represent the most common clinical manifestation although focal neurologic deficits may occur. (From Joynt, Clinical Neurology, 1998, Ch27, pp46-50)
| Descriptor ID |
D020019
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| MeSH Number(s) |
C03.105.250.550 C03.335.190.902.185.550 C10.228.228.205.250.550
|
| Concept/Terms |
Neurocysticercosis- Neurocysticercosis
- Neurocysticercoses
- Cysticercosis, Central Nervous System
- Central Nervous System Cysticercosis
- Cysticercosis, Brain
- Brain Cysticercosis
|
Below are MeSH descriptors whose meaning is more general than "Neurocysticercosis".
Below are MeSH descriptors whose meaning is more specific than "Neurocysticercosis".
This graph shows the total number of publications written about "Neurocysticercosis" by people in this website by year, and whether "Neurocysticercosis" was a major or minor topic of these publications.
To see the data from this visualization as text,
click here.
| Year | Major Topic | Minor Topic | Total |
|---|
| 1999 | 1 | 0 | 1 |
| 2002 | 1 | 0 | 1 |
| 2003 | 2 | 0 | 2 |
| 2006 | 3 | 0 | 3 |
| 2007 | 2 | 0 | 2 |
| 2008 | 3 | 0 | 3 |
| 2009 | 2 | 0 | 2 |
| 2010 | 2 | 0 | 2 |
| 2011 | 1 | 0 | 1 |
| 2012 | 1 | 0 | 1 |
| 2013 | 2 | 0 | 2 |
| 2016 | 1 | 0 | 1 |
| 2017 | 1 | 0 | 1 |
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Below are the most recent publications written about "Neurocysticercosis" by people in Profiles.
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Saito EK, Mehta B, Wang F, Nakamoto B, McMurtray AM. Headaches More Common among Epilepsy Sufferers with Neurocysticercosis than Other Structural Brain Lesions. Hawaii J Med Public Health. 2017 06; 76(6):152-155.
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Mishra PK, Li Q, Munoz LE, Mares CA, Morris EG, Teale JM, Cardona AE. Reduced Leukocyte Infiltration in Absence of Eosinophils Correlates with Decreased Tissue Damage and Disease Susceptibility in ?dblGATA Mice during Murine Neurocysticercosis. PLoS Negl Trop Dis. 2016 06; 10(6):e0004787.
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Mishra PK, Teale JM. Changes in gene expression of pial vessels of the blood brain barrier during murine neurocysticercosis. PLoS Negl Trop Dis. 2013; 7(3):e2099.
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Mishra PK, Morris EG, Garcia JA, Cardona AE, Teale JM. Increased accumulation of regulatory granulocytic myeloid cells in mannose receptor C type 1-deficient mice correlates with protection in a mouse model of neurocysticercosis. Infect Immun. 2013 Apr; 81(4):1052-63.
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Mishra PK, Teale JM. Transcriptome analysis of the ependymal barrier during murine neurocysticercosis. J Neuroinflammation. 2012 Jun 25; 9:141.
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Gundra UM, Mishra BB, Wong K, Teale JM. Increased disease severity of parasite-infected TLR2-/- mice is correlated with decreased central nervous system inflammation and reduced numbers of cells with alternatively activated macrophage phenotypes in a murine model of neurocysticercosis. Infect Immun. 2011 Jul; 79(7):2586-96.
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Mishra BB, Gundra UM, Teale JM. STAT6?/? mice exhibit decreased cells with alternatively activated macrophage phenotypes and enhanced disease severity in murine neurocysticercosis. J Neuroimmunol. 2011 Mar; 232(1-2):26-34.
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Alvarez JI, Mishra BB, Gundra UM, Mishra PK, Teale JM. Mesocestoides corti intracranial infection as a murine model for neurocysticercosis. Parasitology. 2010 Mar; 137(3):359-72.
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Mishra BB, Gundra UM, Wong K, Teale JM. MyD88-deficient mice exhibit decreased parasite-induced immune responses but reduced disease severity in a murine model of neurocysticercosis. Infect Immun. 2009 Dec; 77(12):5369-79.
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Alvarez JI, Krishnamurthy J, Teale JM. Doxycycline treatment decreases morbidity and mortality of murine neurocysticercosis: evidence for reduction of apoptosis and matrix metalloproteinase activity. Am J Pathol. 2009 Aug; 175(2):685-95.