Ischemic Preconditioning, Myocardial
"Ischemic Preconditioning, Myocardial" is a descriptor in the National Library of Medicine's controlled vocabulary thesaurus,
MeSH (Medical Subject Headings). Descriptors are arranged in a hierarchical structure,
which enables searching at various levels of specificity.
Exposure of myocardial tissue to brief, repeated periods of vascular occlusion in order to render the myocardium resistant to the deleterious effects of ISCHEMIA or REPERFUSION. The period of pre-exposure and the number of times the tissue is exposed to ischemia and reperfusion vary, the average being 3 to 5 minutes.
| Descriptor ID |
D019157
|
| MeSH Number(s) |
E02.592.325 E05.516.325
|
| Concept/Terms |
Ischemic Preconditioning, Myocardial- Ischemic Preconditioning, Myocardial
- Myocardial Ischemic Preconditioning
- Preconditioning, Myocardial Ischemic
- Myocardial Preconditioning
- Preconditioning, Myocardial
|
Below are MeSH descriptors whose meaning is more general than "Ischemic Preconditioning, Myocardial".
Below are MeSH descriptors whose meaning is more specific than "Ischemic Preconditioning, Myocardial".
This graph shows the total number of publications written about "Ischemic Preconditioning, Myocardial" by people in this website by year, and whether "Ischemic Preconditioning, Myocardial" was a major or minor topic of these publications.
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| Year | Major Topic | Minor Topic | Total |
|---|
| 2002 | 1 | 0 | 1 |
| 2004 | 1 | 1 | 2 |
| 2007 | 0 | 2 | 2 |
| 2009 | 1 | 0 | 1 |
| 2011 | 1 | 0 | 1 |
| 2014 | 1 | 0 | 1 |
| 2015 | 0 | 1 | 1 |
| 2017 | 1 | 0 | 1 |
| 2019 | 0 | 1 | 1 |
| 2021 | 0 | 1 | 1 |
| 2023 | 1 | 0 | 1 |
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Below are the most recent publications written about "Ischemic Preconditioning, Myocardial" by people in Profiles.
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Knutson AK, Williams AL, Boisvert WA, Shohet RV. HIF in the heart: development, metabolism, ischemia, and atherosclerosis. J Clin Invest. 2021 09 01; 131(17).
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Nu?ez RE, Javadov S, Escobales N. Angiotensin II-preconditioning is associated with increased PKCe/PKCd ratio and prosurvival kinases in mitochondria. Clin Exp Pharmacol Physiol. 2017 Dec; 44(12):1201-1212.
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Nu?ez RE, Castro M, Javadov S, Escobales N. Angiotensin II and ischemic preconditioning synergize to improve mitochondrial function while showing additive effects on ventricular postischemic recovery. J Cardiovasc Pharmacol. 2014 Aug; 64(2):172-9.
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Butler MJ, Chan W, Taylor AJ, Dart AM, Duffy SJ. Management of the no-reflow phenomenon. Pharmacol Ther. 2011 Oct; 132(1):72-85.
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Giorgi C, Agnoletto C, Baldini C, Bononi A, Bonora M, Marchi S, Missiroli S, Patergnani S, Poletti F, Rimessi A, Zavan B, Pinton P. Redox control of protein kinase C: cell- and disease-specific aspects. Antioxid Redox Signal. 2010 Oct 01; 13(7):1051-85.
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Gottlieb RA, Finley KD, Mentzer RM. Cardioprotection requires taking out the trash. Basic Res Cardiol. 2009 Mar; 104(2):169-80.
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Shohet RV, Garcia JA. Keeping the engine primed: HIF factors as key regulators of cardiac metabolism and angiogenesis during ischemia. J Mol Med (Berl). 2007 Dec; 85(12):1309-15.
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Javadov S, Karmazyn M. Mitochondrial permeability transition pore opening as an endpoint to initiate cell death and as a putative target for cardioprotection. Cell Physiol Biochem. 2007; 20(1-4):1-22.
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Qin Q, Yang XM, Cui L, Critz SD, Cohen MV, Browner NC, Lincoln TM, Downey JM. Exogenous NO triggers preconditioning via a cGMP- and mitoKATP-dependent mechanism. Am J Physiol Heart Circ Physiol. 2004 Aug; 287(2):H712-8.
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Halestrap AP, Clarke SJ, Javadov SA. Mitochondrial permeability transition pore opening during myocardial reperfusion--a target for cardioprotection. Cardiovasc Res. 2004 Feb 15; 61(3):372-85.