"Herpesvirus 4, Human" is a descriptor in the National Library of Medicine's controlled vocabulary thesaurus,
MeSH (Medical Subject Headings). Descriptors are arranged in a hierarchical structure,
which enables searching at various levels of specificity.
The type species of LYMPHOCRYPTOVIRUS, subfamily GAMMAHERPESVIRINAE, infecting B-cells in humans. It is thought to be the causative agent of INFECTIOUS MONONUCLEOSIS and is strongly associated with oral hairy leukoplakia (LEUKOPLAKIA, HAIRY;), BURKITT LYMPHOMA; and other malignancies.
| Descriptor ID |
D004854
|
| MeSH Number(s) |
B04.280.210.400.500.450 B04.280.382.400.500.400 B04.613.204.500.500.400
|
| Concept/Terms |
Herpesvirus 4, Human- Herpesvirus 4, Human
- Burkitt Lymphoma Virus
- Lymphoma Virus, Burkitt
- Burkitt's Lymphoma Virus
- Burkitts Lymphoma Virus
- E-B Virus
- E B Virus
- E-B Viruses
- Infectious Mononucleosis Virus
- Infectious Mononucleosis Viruses
- Mononucleosis Virus, Infectious
- Mononucleosis Viruses, Infectious
- Epstein-Barr Virus
- Epstein Barr Virus
- Herpesvirus 4 (gamma), Human
- HHV-4
- Human Herpesvirus 4
- Burkitt Herpesvirus
- Herpesvirus, Burkitt
- EBV
|
Below are MeSH descriptors whose meaning is more general than "Herpesvirus 4, Human".
Below are MeSH descriptors whose meaning is more specific than "Herpesvirus 4, Human".
This graph shows the total number of publications written about "Herpesvirus 4, Human" by people in this website by year, and whether "Herpesvirus 4, Human" was a major or minor topic of these publications.
To see the data from this visualization as text,
click here.
| Year | Major Topic | Minor Topic | Total |
|---|
| 1994 | 1 | 0 | 1 |
| 1995 | 1 | 1 | 2 |
| 1999 | 1 | 0 | 1 |
| 2001 | 0 | 1 | 1 |
| 2002 | 1 | 1 | 2 |
| 2004 | 0 | 1 | 1 |
| 2009 | 0 | 1 | 1 |
| 2010 | 0 | 1 | 1 |
| 2011 | 1 | 0 | 1 |
| 2013 | 1 | 0 | 1 |
| 2015 | 2 | 0 | 2 |
| 2016 | 1 | 0 | 1 |
| 2017 | 1 | 1 | 2 |
| 2018 | 0 | 1 | 1 |
| 2024 | 0 | 1 | 1 |
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Below are the most recent publications written about "Herpesvirus 4, Human" by people in Profiles.
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Kim JY, Min YJ, Lee M-H, An YR, Ashktorab H, Smoot DT, Kwon SW, Lee SK. Ceramide promotes lytic reactivation of Epstein-Barr virus in gastric carcinoma. J Virol. 2024 Feb 20; 98(2):e0177623.
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Kienka T, Varga MG, Caves J, Smith JS, Sivaraman V. Epstein-Barr virus, but not human cytomegalovirus, is associated with a high-grade human papillomavirus-associated cervical lesions among women in North Carolina. J Med Virol. 2019 03; 91(3):450-456.
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Mawson AR, Majumdar S. Malaria, Epstein-Barr virus, vitamin A and Burkitt's lymphoma: Response to Joob and Wiwanitkit. Int J Cancer. 2018 02 15; 142(4):864.
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Mawson AR, Majumdar S. Malaria, Epstein-Barr virus infection and the pathogenesis of Burkitt's lymphoma. Int J Cancer. 2017 11 01; 141(9):1849-1855.
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Mei S, Zhang K. Computational discovery of Epstein-Barr virus targeted human genes and signalling pathways. Sci Rep. 2016 07 29; 6:30612.
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Latham LB, Lee MJ, Lincoln JA, Ji N, Forsthuber TG, Lindsey JW. Antivirus immune activity in multiple sclerosis correlates with MRI activity. Acta Neurol Scand. 2016 Jan; 133(1):17-24.
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Cao S, Moss W, O'Grady T, Concha M, Strong MJ, Wang X, Yu Y, Baddoo M, Zhang K, Fewell C, Lin Z, Dong Y, Flemington EK. New Noncoding Lytic Transcripts Derived from the Epstein-Barr Virus Latency Origin of Replication, oriP, Are Hyperedited, Bind the Paraspeckle Protein, NONO/p54nrb, and Support Viral Lytic Transcription. J Virol. 2015 Jul; 89(14):7120-32.
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Mbulaiteye SM, Pullarkat ST, Nathwani BN, Weiss LM, Rao N, Emmanuel B, Lynch CF, Hernandez B, Neppalli V, Hawes D, Cockburn MG, Kim A, Williams M, Altekruse S, Bhatia K, Goodman MT, Cozen W. Epstein-Barr virus patterns in US Burkitt lymphoma tumors from the SEER residual tissue repository during 1979-2009. APMIS. 2014 Jan; 122(1):5-15.
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Wander K, Shell-Duncan B, Brindle E, O'Connor K. Predictors of delayed-type hypersensitivity to Candida albicans and anti-Epstein-Barr virus antibody among children in Kilimanjaro, Tanzania. Am J Phys Anthropol. 2013 Jun; 151(2):183-90.
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Valent?n-Acevedo A, Sinquett FL, Covey LR. c-Rel deficiency increases caspase-4 expression and leads to ER stress and necrosis in EBV-transformed cells. PLoS One. 2011; 6(10):e25467.