Developmental exposure of mice to dioxin promotes transgenerational testicular inflammation and an increased risk of preterm birth in unexposed mating partners.

Bruner-Tran KL, Ding T, Yeoman KB, Archibong A, Arosh JA, Osteen KG. Developmental exposure of mice to dioxin promotes transgenerational testicular inflammation and an increased risk of preterm birth in unexposed mating partners. PLoS One. 2014; 9(8):e105084.

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subject areas

Animals
Endocrine Disruptors
Environmental Pollutants
Female
Infertility, Male
Inflammation
Male
Mice
Mice, Inbred C57BL
Pregnancy
Premature Birth
Prenatal Exposure Delayed Effects
Spermatozoa
Testis

authors with profiles

Anthony Archibong

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