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Connection

Sabzali Javadov to Mitochondrial Membrane Transport Proteins

This is a "connection" page, showing publications Sabzali Javadov has written about Mitochondrial Membrane Transport Proteins.
  1. Chapa-Dubocq XR, Garcia-Baez JF, Bazil JN, Javadov S. Crosstalk between adenine nucleotide transporter and mitochondrial swelling: experimental and computational approaches. Cell Biol Toxicol. 2023 04; 39(2):435-450.
    View in: PubMed
    Score: 0.820
  2. Javadov S, Jang S, Parodi-Rull?n R, Khuchua Z, Kuznetsov AV. Mitochondrial permeability transition in cardiac ischemia-reperfusion: whether cyclophilin D is a viable target for cardioprotection? Cell Mol Life Sci. 2017 08; 74(15):2795-2813.
    View in: PubMed
    Score: 0.575
  3. Jang S, Lewis TS, Powers C, Khuchua Z, Baines CP, Wipf P, Javadov S. Elucidating Mitochondrial Electron Transport Chain Supercomplexes in the Heart During Ischemia-Reperfusion. Antioxid Redox Signal. 2017 07 01; 27(1):57-69.
    View in: PubMed
    Score: 0.559
  4. Javadov S, Hunter JC, Barreto-Torres G, Parodi-Rullan R. Targeting the mitochondrial permeability transition: cardiac ischemia-reperfusion versus carcinogenesis. Cell Physiol Biochem. 2011; 27(3-4):179-90.
    View in: PubMed
    Score: 0.379
  5. Javadov S, Karmazyn M, Escobales N. Mitochondrial permeability transition pore opening as a promising therapeutic target in cardiac diseases. J Pharmacol Exp Ther. 2009 Sep; 330(3):670-8.
    View in: PubMed
    Score: 0.334
  6. Javadov S, Choi A, Rajapurohitam V, Zeidan A, Basnakian AG, Karmazyn M. NHE-1 inhibition-induced cardioprotection against ischaemia/reperfusion is associated with attenuation of the mitochondrial permeability transition. Cardiovasc Res. 2008 Jan 15; 77(2):416-24.
    View in: PubMed
    Score: 0.298
  7. Javadov S, Karmazyn M. Mitochondrial permeability transition pore opening as an endpoint to initiate cell death and as a putative target for cardioprotection. Cell Physiol Biochem. 2007; 20(1-4):1-22.
    View in: PubMed
    Score: 0.282
  8. Parodi-Rull?n RM, Soto-Prado J, Vega-Lugo J, Chapa-Dubocq X, D?az-Cordero SI, Javadov S. Divergent Effects of Cyclophilin-D Inhibition on the Female Rat Heart: Acute Versus Chronic Post-Myocardial Infarction. Cell Physiol Biochem. 2018; 50(1):288-303.
    View in: PubMed
    Score: 0.159
  9. Chapa-Dubocq X, Makarov V, Javadov S. Simple kinetic model of mitochondrial swelling in cardiac cells. J Cell Physiol. 2018 07; 233(7):5310-5321.
    View in: PubMed
    Score: 0.152
  10. Barreto-Torres G, Hernandez JS, Jang S, Rodr?guez-Mu?oz AR, Torres-Ramos CA, Basnakian AG, Javadov S. The beneficial effects of AMP kinase activation against oxidative stress are associated with prevention of PPARa-cyclophilin D interaction in cardiomyocytes. Am J Physiol Heart Circ Physiol. 2015 Apr 01; 308(7):H749-58.
    View in: PubMed
    Score: 0.123
  11. Escobales N, Nu?ez RE, Jang S, Parodi-Rullan R, Ayala-Pe?a S, Sacher JR, Skoda EM, Wipf P, Frontera W, Javadov S. Mitochondria-targeted ROS scavenger improves post-ischemic recovery of cardiac function and attenuates mitochondrial abnormalities in aged rats. J Mol Cell Cardiol. 2014 Dec; 77:136-46.
    View in: PubMed
    Score: 0.121
  12. Javadov S, Rajapurohitam V, Kilic A, Hunter JC, Zeidan A, Said Faruq N, Escobales N, Karmazyn M. Expression of mitochondrial fusion-fission proteins during post-infarction remodeling: the effect of NHE-1 inhibition. Basic Res Cardiol. 2011 Jan; 106(1):99-109.
    View in: PubMed
    Score: 0.091
  13. Javadov S, Rajapurohitam V, Kilic A, Zeidan A, Choi A, Karmazyn M. Anti-hypertrophic effect of NHE-1 inhibition involves GSK-3beta-dependent attenuation of mitochondrial dysfunction. J Mol Cell Cardiol. 2009 Jun; 46(6):998-1007.
    View in: PubMed
    Score: 0.081
  14. Javadov S, Baetz D, Rajapurohitam V, Zeidan A, Kirshenbaum LA, Karmazyn M. Antihypertrophic effect of Na+/H+ exchanger isoform 1 inhibition is mediated by reduced mitogen-activated protein kinase activation secondary to improved mitochondrial integrity and decreased generation of mitochondrial-derived reactive oxygen species. J Pharmacol Exp Ther. 2006 Jun; 317(3):1036-43.
    View in: PubMed
    Score: 0.067
  15. Halestrap AP, Clarke SJ, Javadov SA. Mitochondrial permeability transition pore opening during myocardial reperfusion--a target for cardioprotection. Cardiovasc Res. 2004 Feb 15; 61(3):372-85.
    View in: PubMed
    Score: 0.058
  16. Javadov SA, Clarke S, Das M, Griffiths EJ, Lim KH, Halestrap AP. Ischaemic preconditioning inhibits opening of mitochondrial permeability transition pores in the reperfused rat heart. J Physiol. 2003 Jun 01; 549(Pt 2):513-24.
    View in: PubMed
    Score: 0.054
  17. Nu?ez RE, Javadov S, Escobales N. Angiotensin II-preconditioning is associated with increased PKCe/PKCd ratio and prosurvival kinases in mitochondria. Clin Exp Pharmacol Physiol. 2017 Dec; 44(12):1201-1212.
    View in: PubMed
    Score: 0.037
  18. Kuznetsov AV, Javadov S, Saks V, Margreiter R, Grimm M. Synchronism in mitochondrial ROS flashes, membrane depolarization and calcium sparks in human carcinoma cells. Biochim Biophys Acta Bioenerg. 2017 Jun; 1858(6):418-431.
    View in: PubMed
    Score: 0.036
  19. Nu?ez RE, Castro M, Javadov S, Escobales N. Angiotensin II and ischemic preconditioning synergize to improve mitochondrial function while showing additive effects on ventricular postischemic recovery. J Cardiovasc Pharmacol. 2014 Aug; 64(2):172-9.
    View in: PubMed
    Score: 0.030
Connection Strength

The connection strength for concepts is the sum of the scores for each matching publication.

Publication scores are based on many factors, including how long ago they were written and whether the person is a first or senior author.
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