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Expression profile and role of EphrinA1 ligand after spinal cord injury.
Blockade of P2 nucleotide receptors after spinal cord injury reduced the gliotic response and spared tissue.
P2Y2 receptor expression is altered in rats after spinal cord injury.
Expression and activation of ephexin is altered after spinal cord injury.
Cocaine sensitization increases I h current channel subunit 2 (HCN2) protein expression in structures of the mesocorticolimbic system.
Docosahexaenoic acid pretreatment confers protection and functional improvements after acute spinal cord injury in adult rats.
Expression profile of flotillin-2 and its pathophysiological role after spinal cord injury.
Tamoxifen and estradiol improved locomotor function and increased spared tissue in rats after spinal cord injury: their antioxidant effect and role of estrogen receptor alpha.
Fatty Acid Binding Protein 5 Modulates Docosahexaenoic Acid-Induced Recovery in Rats Undergoing Spinal Cord Injury.
Tamoxifen Administration Immediately or 24 Hours after Spinal Cord Injury Improves Locomotor Recovery and Reduces Secondary Damage in Female Rats.
Continuous tamoxifen delivery improves locomotor recovery 6h after spinal cord injury by neuronal and glial mechanisms in male rats.
Upregulation of EphA receptor expression in the injured adult rat spinal cord.
Transection of the adult rat spinal cord upregulates EphB3 receptor and ligand expression.
Blocking EphA4 upregulation after spinal cord injury results in enhanced chronic pain.
Inhibition of EphA7 up-regulation after spinal cord injury reduces apoptosis and promotes locomotor recovery.
Reduction of EphA4 receptor expression after spinal cord injury does not induce axonal regeneration or return of tcMMEP response.
Upregulation of EphA3 receptor after spinal cord injury.
EphB3 receptor and ligand expression in the adult rat brain.
Molecular, anatomical, physiological, and behavioral studies of rats treated with buprenorphine after spinal cord injury.
Rats Sprague Dawley
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