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Matsui, Takashi
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Item TypeName
Academic Article mTOR attenuates the inflammatory response in cardiomyocytes and prevents cardiac dysfunction in pathological hypertrophy.
Academic Article Cardiac mTOR protects the heart against ischemia-reperfusion injury.
Academic Article Deep sequence analysis of gene expression identifies osteopontin as a downstream effector of integrin-linked kinase (ILK) in cardiac-specific ILK knockout mice.
Academic Article Cardiac mTOR rescues the detrimental effects of diet-induced obesity in the heart after ischemia-reperfusion.
Academic Article The mTOR Signaling Pathway in Myocardial Dysfunction in Type 2 Diabetes Mellitus.
Academic Article Protective effects of the mechanistic target of rapamycin against excess iron and ferroptosis in cardiomyocytes.
Academic Article Akt and PI 3-kinase signaling in cardiomyocyte hypertrophy and survival.
Academic Article Phenotypic spectrum caused by transgenic overexpression of activated Akt in the heart.
Academic Article Endothelial IKK beta signaling is required for monocyte adhesion under laminar flow conditions.
Academic Article Transcriptional effects of chronic Akt activation in the heart.
Academic Article Strategic advantages of insulin-like growth factor-I expression for cardioprotection.
Academic Article Convergent signal transduction pathways controlling cardiomyocyte survival and function: the role of PI 3-kinase and Akt.
Academic Article Serum and glucocorticoid-responsive kinase-1 regulates cardiomyocyte survival and hypertrophic response.
Academic Article PI3K rescues the detrimental effects of chronic Akt activation in the heart during ischemia/reperfusion injury.
Grant ROLE OF PI 3-KINASE AND AKT IN CARDIOMYOCYTE APOPTOSIS
Academic Article Akt activation preserves cardiac function and prevents injury after transient cardiac ischemia in vivo.
Academic Article Adenoviral gene transfer of activated phosphatidylinositol 3'-kinase and Akt inhibits apoptosis of hypoxic cardiomyocytes in vitro.
Academic Article mTOR-mediated calcium transients affect cardiac function in ex vivo ischemia-reperfusion injury.
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